International Meeting for Autism Research: The Dog as a Genetic Model to Investigate Overlap in Social, Attention, and Activity Behaviors

The Dog as a Genetic Model to Investigate Overlap in Social, Attention, and Activity Behaviors

Friday, May 21, 2010
Franklin Hall B Level 4 (Philadelphia Marriott Downtown)
2:00 PM
L. Lit , Neurology & Psychiatry and Behavioral Sciences, University of California at Davis, Sacramento, CA
D. Bannasch , Population Health and Reproduction, University of California at Davis, Davis, CA
J. Schweitzer , Psychiatry and Behavioral Sciences, University of California at Davis, Sacramento, CA
Background:

Currently, there are limited animal models to provide insight into genetic underpinnings of social behaviors and the often comorbid inattention/hyperactivity behaviors observed in autism spectrum disorders (ASDs).  Social interactive behaviors between domestic dogs (Canis familiaris) and humans have been well documented experimentally.  Additionally, owner reports of social and attention/activity behaviors have been used to characterize variability in naturally occurring dog behaviors that are homologous to those relevant to ASDs.  It has been hypothesized that a limited subset of wild canids originally displayed the social characteristics necessary to interact with humans, ultimately resulting in domestication.

Objectives:

Determine whether there would be an overlapping set of genes associated with social behaviors and attention/activity behaviors in domestic dogs.

Methods:

We asked owners of 14 dogs (1 Basset Hound, 1 Black Russian Terrier, 2 Dalmatians, 1 Petit Basset Griffon Vendeen, 7 Nova Scotia Duck Tolling Retrievers, and 2 mixed breed dogs) to complete surveys comprising owner report of social and attention/activity behaviors observed in their dogs.  Social behaviors subscales 1) initiation of reciprocal social behaviors (INIT), 2) response to social interactions with humans (RSPNS), and 3) communication with humans (COMM); as well as attention (ATT) and activity (ACT) subscales were derived from responses using factor analysis. Genome-wide SNP data for these dogs was generated using 127K Affymetrix canine single nucleotide polymorphism (SNP) arrays.  Using subscales as input, we performed genome-wide quantitative trait association to determine SNPs associated with each subscale.  

Results:

Overlapping SNPs associated with both a social subscale and either an attention or activity subscale were identified.  Most SNPs corresponded to genes or regions homologous to those in humans associated with autism, ADHD, and schizophrenia.

Conclusions:

These findings contribute to the validity of the domestic dog as an animal model to investigate overlap in genetic backgrounds of social and attention/activity behaviors.

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